Metabolic syndrome and abdominal obesity increase health risks. Both disorders include a cluster of risk factors for cardiovascular disease, type 2 diabetes, and other health issues.
Abdominal obesity is extra fat surrounding visceral organs such the liver, pancreas, and intestines.
Metabolically active visceral fat releases inflammatory chemicals. Insulin resistance, which raises blood sugar, can ensue.
Abdominal obesity causes insulin resistance, which raises type 2 diabetes risk. Insulin resistance reduces cell glucose absorption, raising blood sugar.
Increased abdominal fat changes hormone and cytokine synthesis, including adiponectin and leptin. Hormonal imbalance can cause inflammation and metabolic malfunction.
Abdominal obesity increases the risk of hypertension, atherosclerosis, and coronary artery disease.
Depending on gender and population, metabolic syndrome requires a waist circumference over specified thresholds.
Fasting blood glucose levels over 100 mg/dL indicate metabolism problems and lead to metabolic syndrome.